P12

Oncogenic BRAF-V600E drives immune escape via prostaglandin E2/oncostatin M production in melanoma

Project Summary

Project P12 aims to uncover the mechanisms by which the BRAF-V600E mutation in malignant melanoma promotes immunosuppression within the tumour microenvironment, focusing specifically on the roles of Oncostatin M and prostaglandin E2 in modulating myeloid cells. To dissect this regulatory axis, we will employ in vitro co-culture systems as well as orthotopic and metastatic melanoma mouse models. As a translational approach, we will investigate the therapeutic potential of combining anti-OSMR antibodies with BRAF/MEK inhibitors and anti-PD-1 immunotherapy and validate our findings in human melanoma tissue samples.

  • P01Blaeschke / Zeiser
  • P02Greten
  • P03Köhler / Briquez
  • P05Apostolova / Kierdorf
  • P07Prinz
  • P08Frew
  • P10Heikenwälder / Hofmann
  • P12Duyster / Kolter
  • P14Brummer
  • P15Minguet
  • P17Ruess / Bengsch
  • P18Hoefflin / Sevenich
  • P19Hofmann / Röhlen
  • P20Metzger
  • P21Böttcher
  • P22Feuchtinger / Maas-Bauer
  • P23Ingelfinger / Sevenich
  • P24Vinnakota / Wertheimer
  • P25Pahl / Rizzi
  • S1Börries / Köttgen / Schell
  • S2Reichardt / Talvard-Balland
  • INFBinder
  • ZZeiser
  • IRTGBörries
  • P16EErlacher
  • P06EGroß
  • P04EIllert
  • P13ENyström / Kiritsi
  • P05 1.FPCabezas-Wallscheid
  • S01 1.FPSchilling